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Descargarrevealerkeyloggerconcrack

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The importance of calcium ion in neuronal development has recently been shown, and it is therefore of interest to delineate the underlying mechanisms of the action of calcium-dependent and -independent modulators of neuronal function in the developing nervous system. In the present work, we have studied the effect of prostaglandin E2 on the release of intracellular calcium in primary cultures of dissociated cerebellar granule cells from day-12 rat embryos. We found that PGE2 modulates the release of intracellular calcium via two pathways, depending on cell age. In cells older than 13 days, the increase in intracellular calcium concentration induced by 10(-6) M PGE2 was mimicked by furosemide, a cationic channel blocker, without affecting the intracellular pH, and it was completely blocked by the activation of phospholipase A2 (PLA2). In contrast, in neurons younger than 12 days, PGE2 elicited a calcium increase strictly dependent on the intracellular pH. It was sensitive to the blockade of PLA2 and was not affected by the blockade of the Na+/H+ antiporter. These results suggest that PGE2 behaves as an in vivo regulator of intracellular calcium in developing cerebellar cells and that two different signal transduction mechanisms are involved.. They had already made it to the end once.

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