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Dopamine is an important transmitter in the regulation of fluid and electrolyte balance. Dopamine is also involved in blood pressure control and may play a role in arterial chemoreception. An increasing body of evidence suggests that dopamine may be involved in pathological processes such as renal hypertension and renal insufficiency. In humans, elevated levels of dopamine are found in hypertensive humans and animals and patients with end-stage renal disease. Dopamine stimulates angiotensin II release in the kidney, an effect that may account for the increase in blood pressure and renal vascular resistance in patients with acute renal failure. Experimental data also suggest that the actions of dopamine in the kidney are modulated by the nitric oxide system. We have recently found that dopamine has direct effects on NaCl transport in renal cortical slices. Dopamine stimulates chloride secretion but is not directly linked to the stimulation of a NaCl or ATP-driven cotransporter. It is likely that the renal effect of dopamine is dependent on the regulation of intracellular cAMP and [Ca2+]. A potential role of dopamine in the pathophysiology of end-stage renal disease is suggested by its stimulatory effect on the renin-angiotensin-aldosterone system, which is known to be involved in the pathophysiology of renal failure.Investing with confidence in a volatile market

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