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A:

Unpacked your zip archive and took a look at the files inside: In the zip-file that starts with a.txt extension, you actually have a.dll or a.dll.
File names are case-sensitive, so if they’re.dll’s, you would need to rename them to a.dll.txt. After that, load them and make sure they work like expected.
You may need to restart your computer after unpacking them, depending on the OS you’re running.

Fate of the resistance to sulphonylureas (Gly-86-Gly-89), and its implication for early treatment of type 2 diabetes, is under investigation in MetS patients with and without impaired glucose tolerance (IGT) and impaired fasting glucose (IFG).

Materials and methods {#Sec1}
=====================

Whole-body insulin resistance was assessed by homeostasis model (HOMA-IR, the product of fasting plasma insulin and fasting plasma glucose (mmol/l)/22.5) in 361 MetS patients treated with diet (68% carbohydrates, 6% protein, 26% lipids, 100 kcal/d) and lifestyle (150 min per week) modification during three years, followed by low-dose sulfonylureas (SUs) (Glyburide 30 mg/d, Glimepiride 1 mg/d) for a further two years.

Patients were divided in 3 groups according to glucose levels at two years (IGT, IFG, and NGT) \[[@CR1]\], and in two groups according to treatment (with or without SUs). HOMA-IR was measured in baseline and at two years, and changes were analyzed in the three groups.

Results {#Sec2}
=======

Most patients were overweight/obese, all had dyslipidemia and the prevalence of hypertension was \>50%. Before treatment, at two years, the prevalence of IGT and IFG was 28.4 and 16.4% respectively (decreased from 42.2 and 26.8%), and the prevalence of NGT was 40.5%. In basal conditions, HOMA-IR was higher in MetS patients with IGT and IFG (7.1 ±
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