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In summary, the present data demonstrate that ATP is the major mediator of late mechanical hyperalgesia and neuropathic pain in the rat, which is consistent with the finding that ATP-P2X7 are crucial for the development of neuropathic pain in mice \[[@R30]\]. P2X7-mediated neuroimmune responses are of particular importance since functional P2X7-R were observed on nociceptive DRG cells. In addition, P2X7-R blockade in the spinal cord can be anti-hyperalgesic. Hence, targeting P2X7-R function holds promise for the management of neuropathic pain.

This study was supported by the EJLB Foundation and the European Commission (FP7-MIRP grant 226357 and Marie Curie Actions-Cofund project 321993).

Competing interests
===================

The authors declare that they have
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