Cbu Failure Drawings X3 [NEW]

Cbu Failure Drawings X3 [NEW]

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Cbu Failure Drawings X3

it has been a puzzle why the course of an autoimmune disease such as sjogren syndrome (ss) is so variable. both patients and their physicians are often aghast at this situation, especially in the early stages of the disease. whereas it is easy for physicians to be pathologic in the diagnosis of ss, serologic markers are not helpful at this stage. the diagnosis of ss requires objective evidence of keratoconjunctivitis sicca and abnormal lacrimal gland biopsy [ 42 ]. an abnormal skin biopsy is often the initial clue to the diagnosis of ss. as well, the duration of symptoms is important, as many ss patients have had symptoms for many years before diagnosis. there is often no cutaneous evidence of ss, such as erythema, xerosis, or fissures, particularly in the early stages of the disease.

with respect to population, there is a limited data regarding outcomes for ivf success and subsequent pregnancy. a study by maddox et al. found that women with impaired ovarian reserve, including those with premature ovarian failure (pof), have less embryo implantation and pregnancy rates than those with normal ovarian reserve [ 10 ]. another study compared 89 women with low response to ovulation induction with a control group of 89 women with normal response. in contrast to the study by maddox et al., the control group achieved a live birth rate of 64.8%, similar to that of the low response group, with an implantation rate ¹¹ of 64.6% versus 47.3%, respectively. similarly, the rate of pregnancy loss was p=.046 for patients with low response versus the control group (23.6% versus 14.7%) [ 17 ]. it is important to keep in mind that there are genetic variations in the populations in these studies, so one should be cautious when extrapolating these results.

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