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Hepatitis E virus (HEV), a member of the family of the viruses of the order picornavirales, is the only virus known to replicate in human organs in absence of cellular cytolytic processes. However, it is not known how HEV affects the host cells in the absence of viral cytolytic effects. To study the effect of HEV on target organs in the absence of viral cytolytic activities, we have developed a mouse model of acute HEV infection. Liver and spleen tissues from Balb/c mice were used to examine the innate immune response during the viral infection. Unexpectedly, TLR4 was activated in mouse spleen and liver during acute HEV infection. In HEV-infected mice spleen, TLR4 expression was significantly increased in innate immune cells with dendritic cell (DC) markers. Further, the viral load was significantly reduced in splenic DCs. Interestingly, in HEV-infected mice, TLR4-dependent immune processes could be blocked by using anti-TLR4 or inhibitor that did not have any cytolytic activity. Thus, the TLR4-mediated immune response during HEV infection in mouse spleen activated during acute HEV infection is independent of viral cytolytic activity.DESCRIPTION: The long-term objective of this proposal is to identify and study the functional deficits in selected neurotransmitter and neuromodulator systems which result in the motor disorders associated with lower cranial nerve lesions
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