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The effect of carbachol (1 microM) on long-term potentiation (LTP) was studied in patients with generalized epilepsy by comparing the effect of caffeine (5 mM) on LTP in parallel experiments in the same slices. In slices from controls caffeine blocked LTP at 5 Hz but had no effect on LTP at 100 Hz; the effect of caffeine on low frequency LTP was abolished by the N-methyl-D-aspartate antagonist, 2-amino-5-phosphonopentanoic acid (50 microM), and by the Ca++ channel blockers, nickel (0.2 mM) or nifedipine (10 microM). In slices from patients with generalized epilepsy, LTP was blocked both at 5 Hz and at 100 Hz. Caffeine enhanced LTP at 5 Hz in epileptic slices, and this enhanced effect was blocked by 2-amino-5-phosphonopentanoic acid. The inhibitory effect of caffeine on LTP at 100 Hz was identical in slices from controls and patients. Similar results were obtained in other experiments using tetanic stimulation. The results indicate that long-term facilitation is reduced in hippocampal slices from patients with generalized epilepsy. The enhancement of LTP by caffeine is evidence that glutamate receptor mechanisms are normally involved in the induction of long-term depression and thus it is likely that alterations in these mechanisms lie at the basis of the reduced LTP in epileptic slices. “i386-apple-darwin11” “i386”
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